The endocannabinoid anandamide (AEA) is an antinociceptive lipid that’s inactivated through cellular uptake and subsequent catabolism by fatty acid amide hydrolase (FAAH). inhibitors mirrored their affinities for FABP5 even though binding to FABP7 and FABP3 had not been a predictor of effectiveness. The antinociceptive ramifications of FABP inhibitors had been mediated by cannabinoid receptor 1… Continue reading The endocannabinoid anandamide (AEA) is an antinociceptive lipid that’s inactivated through